GPR35 agonists (YE120, zaprinast, and pamoic acid) promoted wound restore in a very concentration-dependent fashion independently of cell proliferation, Whilst a particular GPR35 antagonist CID2745687, forskolin, and pertussis toxin reversed the YE120-induced effect. YE120 amplified the mRNA expression of fibronectin and its receptor integrin α5, and ERK1/2 phosphorylation, but these https://stattic65432.blogripley.com/28999616/the-ultimate-guide-to-ye-120